Source: Experimental and Toxicologic Pathology. Unidades: FZEA, FMVZ
Subjects: BIOLOGIA CELULAR, CONEXINAS, APOPTOSE, CAMUNDONGOS
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FUKUMASU, Heidge et al. Transient disruption of liver gap junctional intercellular communication and induction of apoptosis after administration of 1,4-bis[2-(3,5 dichloropyridyloxy)] benzene in mice. Experimental and Toxicologic Pathology, v. 62, n. 5, p. 525-531, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.etp.2009.07.001. Acesso em: 03 maio 2024.APA
Fukumasu, H., Sanches, D. S., Silva, T. C., Ward, J. M., & Dagli, M. L. Z. (2010). Transient disruption of liver gap junctional intercellular communication and induction of apoptosis after administration of 1,4-bis[2-(3,5 dichloropyridyloxy)] benzene in mice. Experimental and Toxicologic Pathology, 62( 5), 525-531. doi:10.1016/j.etp.2009.07.001NLM
Fukumasu H, Sanches DS, Silva TC, Ward JM, Dagli MLZ. Transient disruption of liver gap junctional intercellular communication and induction of apoptosis after administration of 1,4-bis[2-(3,5 dichloropyridyloxy)] benzene in mice [Internet]. Experimental and Toxicologic Pathology. 2010 ; 62( 5): 525-531.[citado 2024 maio 03 ] Available from: https://doi.org/10.1016/j.etp.2009.07.001Vancouver
Fukumasu H, Sanches DS, Silva TC, Ward JM, Dagli MLZ. Transient disruption of liver gap junctional intercellular communication and induction of apoptosis after administration of 1,4-bis[2-(3,5 dichloropyridyloxy)] benzene in mice [Internet]. Experimental and Toxicologic Pathology. 2010 ; 62( 5): 525-531.[citado 2024 maio 03 ] Available from: https://doi.org/10.1016/j.etp.2009.07.001
